C02-H stimuli and neural muscular drive to ventilation during dynamic exercise- comparison of stimuli at constant levels of ventilation
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| Deutscher übersetzter Titel: | C02-H-Reiz und neuromuskulaerer Anreiz zur Ventilation waehrend dynamischer Muskelarbeit. Vergleich der Stimuli bei konstant gehaltener Ventilation |
|---|---|
| Autor: | Essfeld, D.; Stegemann, Jürgen |
| Erschienen in: | International journal of sports medicine |
| Veröffentlicht: | 4 (1983), 4, S. 215-222, Lit. |
| Format: | Literatur (SPOLIT) |
| Publikationstyp: | Zeitschriftenartikel |
| Medienart: | Gedruckte Ressource Elektronische Ressource (online) |
| Sprache: | Englisch |
| ISSN: | 0172-4622, 1439-3964 |
| DOI: | 10.1055/s-2008-1026037 |
| Schlagworte: | |
| Online Zugang: | |
| Erfassungsnummer: | PU198403020725 |
| Quelle: | BISp |
Abstract
In exercising man, the ventilatory responses to C02-H+ stimuli and neural muscular drives were compared at constant ventilation (VE). For that purpose, a small incraese of the C02-H+ stimulus in exercise was to be counter-balanced by work load reductions in such a way that the magnitude of ventilation remained unchanged. Only in metabolic acidosis could the additional C02 stimulus be compensated by work load reduction. This compensation was due to the concomitant decrease of acidosis. Below the 2 mmol/l (La) a threshould, decrements of work load, V02, and VC02 showed no effect on VE, when PETC02 and PET02 were regulated at constant levels. The results show that neural muscular drives cannot decrease ventilation against a background of constant arterial feedback stimuli. Transient decreases of the C02-H+ stimulus seem to be necessary to readjust the ventilation to a decreased C02 flow to the lungs. It is suggested that the overall effect of decreasing C02 is to inhibit the respiratory centers and that positive ventilatory effects of CO2 are the results of a disinhibitory influence. Verf.-Referat